Faith Integration Case Study 3: The Biological Basis for Disease in Adults and Children

Faith Integration Case Study 3
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McCance & Huether (2018) deems paraplegia as a sensory or motor function impairment of the thigh, hip leg, ankle, or foot. Inferior limb pelvic limb and ankle also identified as lower extremities.Paraplegia is caused by an innate condition or spinal cord injury that affects the spinal canal neural brain elements. It mainly affects the pelvic region, legs, trunk resulting in loss of movement. The authors further explain that paraplegia has diverse causes such as severe spinal cord injury, motor neuron disease, accidents, spina bifida, prolonged diseases, alcohol addiction and cancerous cell growth.
Paraplegia pathophysiology seeks to explain the mechanisms or processes where the undesired or abnormal condition develops and progresses (Eldahan & Rabchevsky 2018). It can also imply the functional changes linked with paraplegia disease. Spinal cord injury pathophysiology is categorized into compression or acute impact. According to Denis, Feldman, Thompson & Mac-Thiong (2018) acute impact injury is a spinal cord concussion. The acute impact injury prompts an unforeseen chain of events targeting the grey matter thus resulting in hemorrhagic necrosis. The grey matter hypoperfusion is the initial event. Reperfusion injury and intracellular calcium increment triggers cellular injury and occurs prior and post-injury. The necrosis extent is contingent on the initial vigor of trauma though it also incorporates blood flow and perfusion pressure, concomitant compression and administration of pharmacological agents. McCance & Huether (2018) argues that the prevention of these cascading events is subject to mechanisms that occur in the initial stages. A mass impinging on the spinal cord triggering accelerated parenchymal pressure causes spinal cord compression. The tissue responds by demyelination, gliosis and axonal loss. This only occurs in the white matter while preservation occurs in gray matter structures. The critical compression degree results in microvasculature venous side collapsing amounting to vasogenic edema. A rapid progression of disfunction is attributable to parenchymal pressure exacerbated by vasogenic edema. The treatment of the compression majors on offending mass removal (Gill et.al, 2018)
Addressing the patient’s spiritual needs was simpler. The spiritual needs were very personal implicating tha...